March 2008 NCLEX support group

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A thread for all those in March who are planning to take the NCLEX offering both support and any hints or tips that help

Good luck to all :D

I took NCLEX practice test at Kaplan center today and I got 65% score and I will take my NCLEX on March 17 .I barely passed, but I still worried for my real exam, beacause I am so close to gray zone:uhoh3:

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Is anybody using this website to study?????

I think I got the address from the thread somewhere here. If you are using the site to study, do you think all the information that's given there is correct? Like I was going through the ACUTE RENAL FAILURE information.

According to the slide show this is what it said about the diuretic phase for ARF:

"slide 51: Acute renal failure Assessment findings: The Three Phases of Acute Renal Failure 2. Diuretic phase  Diuresis may occur (output 3—5 liters/day) due to partially regenerated tubule’s inability to concentrate urine  Duration: 2—3 weeks; manifested by hyponatremia, hypokalemia, and hypovolemia  Diagnostic tests: BUN and creatinine slightly elevated"

Electrolytes during Diuretic phase a/c to the site is: Low Na, Low K

But a/c to ATI book, it's High Na, High K

And, I would think ATI is correct for Diuretic phase.

So how trustworthy are the slides now? You know I thought it was very helpful slide shows. Now, I don't know what to make of it. I picked this error out because it wasn't making sense to me, how can sodium value be in same direction (that is low) for both oliguric and diuretic phase? I must have studied so many things wrong all along then. These slide shows were so helpful, well I haven't done many of them, but the ones I scanned :s . What's this?

Someone, please reply.

And, in oliguric phase for ARF the elecrtolyte values are high for both sodium and Potassium right??? I would think it to be high. Please reply. Your responses will be very much appreciated.

Sorry guys, sometimes I sit back and think I must be spreading so much negative energy here. I'm always whining :) Sorry. I'm sending positive energy to all of us. U know they say, those who have no one, they have God. So, let's make it happen guys. Cheers to all of us.

P.s When u guys do study mode in Saunders: How do you use bookmarks? What are they there for? Hhow do you go back to the questions you bookmarked? Please reply, if someone knows on this. Your help will be really appreciated. Take care all.

And thank you to guys, who passed their RN tests and coming back and asking us if we need help. That really means a lot. Congratulations again. Only, you know what it is trying to reach for the hope, like struggling in the avalanche and all. But, we will do it.

Happy international women's day to all. Sorry this got long.

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http://www.slideshare.net/nclexvideo...urinary-system

Please, do go thru the website, if anyone has time. Or if someone has used it to study and let me know, if I should stop studying thru it. Please :). Thank you so much

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So for Acute Renal failure:

3 phases: Oliguric phase, Diuretic phase and recovery phase

Oliguric phase: Fluid retention

urine output less than 400 ml/day

Elevated BUN, creatinine, Potassium, phosphorus

Metabolic acidosis

N & V, metallic taste in mouth.

Intervention: low sodium, potassium, protein diet

High calorie diet

Diuretic phase:

3-5 L/day

Elevated BUN, creatinine, potassium, sodium

Intervention: fluid replacement: output + 500 mL/day

High potassium, sodium diet

low protein diet

Recovery phase: monitor s/s CRF - chornic renal failure

Question:

What is the value of sodium in Oliguric phase?

Elevated also?

Thanks

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Oliguric phase - low sodium?

Low sodium that's y, ur urine output is low ????

Diuretic phase - high sodium, so urine output is high.

What is the rational behind Low urine output for oliguric phase?

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Function of ADH:

"Antidiuretic hormone increases teh permeability to water of the distal convoluted tubules and collecting ducts" Can someone please explain this in simple words? Thank you

ADH = Less Pee means less urine formed in tubules coz any solutes and fluids that are not reabsorbed from tubules become urine. So this means more fluids and electrolytes are reabsorbed from tubules when ADH in effect. This means more water able to flow from the tubules and collecting ducts through the semipermeable membrane into the blood?

Am I understanding it correct? Someone please help

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hello everyone!! just a thought..... to all those who passed the nclex & who ONLY did kaplan q bank & did NOT read any content, what was your average? (i'm currently doing the kaplan q bank & i'm also curious what a "successful" percentage is.) as i go through this site, i'm amazed as to how ppl pass the nclex w/ out reading & just doing practice questions. honestly, how in the world is it possible to not know the content & pass? i knoe many ppl believe that since the nclex is so out there, it's pointless to study content & the focus is just on knowing how to take the exam. i've been given the advice of not studying & just doing practice questions & reading the rationales so many times that at this point, they make me feel like i'm wasting my time reading the content! i seriously thought that passing the nclex would be knowing content + knowing how to take the exam. and as my test date comes near, i think i'm just feeling a bit frustrated ' cuz i don't even know if i'm studying "correctly." i started off wanting to cover content (reading, taking end of chapter test, etc... ) & then working on online practice tests. but right now, i just don't know if i'm on the right path.... *sigh* :o

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Sorry I havent got any of my books with me, so I'm using the web for basis:

Serum creatinine; in prerenal failure the ratio of urinary to plasma creatinine is high- > 40. The urinary sodium concentration is low -

http://www.patient.co.uk/showdoc/40000105/

As we have said, a normally functioning kidney is able to conserve salt and water. A sensitive indicator of tubular function is sodium handling because the ability of an injured tubule to reabsorb sodium is impaired, whereas an intact tubule can maintain this reabsorbtive capacity in the face of a hemodynamic stress. With a prerenal insult, the urine sodium should be less than 20, and the calculated fractional excretion of sodium should be less than 1%.

http://www.ccmtutorials.com/renal/pathphys/page_06.htm

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function of adh:

"antidiuretic hormone increases teh permeability to water of the distal convoluted tubules and collecting ducts" can someone please explain this in simple words? thank you

adh = less pee means less urine formed in tubules coz any solutes and fluids that are not reabsorbed from tubules become urine. so this means more fluids and electrolytes are reabsorbed from tubules when adh in effect. this means more water able to flow from the tubules and collecting ducts through the semipermeable membrane into the blood?

am i understanding it correct? someone please help

yeah u got it pretty much. adh binds to the receptors of cells at the ducts of kidney, making reabsorption of water back into the circulation possible. permeability is like, think maybe like a gate. if there's adh (key) that binds to the cell receptors (keyhole), water can get through and it gets reabsorbed. no adh, water cant get through, and it becomes urine.

ok am i making sense too? 'cuz i dunno. hahaha! :lol2:

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hello!i am taking the exam on the 24th.I feel ambivalent at this time.i dont know if this is healthy for me or if this is an indicator of me being too confident or too numb to face that the day is near.there is much uncertainty sorrounding the exam but i know 1 thing is for sure.God has a plan for all of US.We need just to believe and as we believe we'll very well be on our way to what is in store US.Goodluck to all of US and more power!God be with US all!!!:yeah:

P.S this site had helped me a lot to settle down and be more aground with the realities of NCLEX!with this a bunch of Thank Yous to those who founded it!:yeah:

Please include me in your prayers!I'll definitely be praying for all of us march test takers!it is a pleasure to be included in this group.I wish all of you guys and gals the best in life.Again God be with us ALL!!:yeah:

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just wondering, in validating the placement of a gastric tube is it true that aspirating the tube for gastric contents is better than obtaining an x-ray?tanx a lot!god bless!!!:D

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Specializes in Cardiac/Telemetry.
just wondering, in validating the placement of a gastric tube is it true that aspirating the tube for gastric contents is better than obtaining an x-ray?tanx a lot!god bless!!!:D

Actually, two of the first things you do is insert 10cc's of air into the NG tube and auscultate. You should hear a "whoosh". Then, you aspirate 10cc's of gastric contents. Sometimes, you check the pH to make sure that it is between 4-5 pH and that it is gastric contents. After all of that, then you check with an X-Ray. :)

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