Test Your Neonatal Critical Care Knowledge - page 2
So, by popular demand..... here is a thread dedicated to questions posed to help increase one's knowledge base in regards to the acute care NICU population. If anyone desperately wants a separate... Read More
0Mar 4, '10 by littleneoRNQuote from SteveNNPSteve, do you mean the opposite? That kids with head sparing have weight and length that plots small but the head plots normal (or least higher on the scale than weight/length)?Head-sparing, or asymmetrical IUGR is reassuring. Babies with this have a head circ <10% pct, but the length and weight plot normally. This form of IUGR occurred later in pregnancy and the brain and head were spared from lack of oxygen/nutrients.
1Mar 4, '10 by SteveNNP, MSN, NPQuote from littleneoRNYou're right! See what happens when I post before coffee?Steve, do you mean the opposite? That kids with head sparing have weight and length that plots small but the head plots normal (or least higher on the scale than weight/length)?
0Mar 4, '10 by TiffyRN, ASN, BSN, RNOK, I was just going to comment how much I was enjoying this. I am woefully lacking in knowledge of maternal factors influencing infants. I recognize this as a flaw that needs improving. The section on end-diastolic flow was so so helpful, I see that all the time on our infant hx section. It makes so much sense now!
One thing I remember hearing from one of our neonatologists was that though generally asymmetric IUGR is not as ominous as symmetric for neuro outcomes, they (the asymmetric infants) are still at increased risk compared to other preemies for PVL. I don't know why, but if it's true, I bet someone here knows.
0Mar 5, '10 by chareQuote from stevennptransposition of the great arteries (tga). in tga the aorta exits the right ventricle, and the pulmonary artery arises exits left ventricle.question #3:
name the cyanotic congenital heart defect most commonly diagnosed within the first week, and briefly describe the path of circulation in the heart.
in the immediate post natal period the pressures in pulmonary circulation remain roughly the same as the pressures in the peripheral circulation and this allows the structures associated with the fetal circulation (ductus arteriosus and foramen ovale) remain open. this initially allows oxygenated blood to shunt to the right side of the heart. as these structures begin to close, this shunting will either stop, or change direction and become a right to left shunt, further complicating the problem by overloading the pulmonary circulation leading to congestive heart failure.
eventually the infant will require an arterial switch procedure, however prior to that he or she will likely be started on prostaglandins to maintain the ductus arteriosus and undergo a rashkind procedure (balloon atrial septostomy) to maintain and/or enlarge the foramen ovale.
0Mar 5, '10 by NotReady4PrimeTime, RN Senior ModeratorMy son has a corrected TGA. He was born in the Senning-Mustard era so has a functionally backwards heart, with intra-artial baffles redirecting blood flow. Fortunately he has managed to avoid the usual complications and morbidities associated with these procedures, the atrial dysrhythmias, the vegetations and emboli, the baffle and vascular stenosis, the RV failure that are so common in adolescent and adult patients. His heart looks very different on echo; it's almost vertical, rotated a bit posteriorly and to the left with long, narrow ventricles, giving the tech some difficulty in getting good images. The cardiologist told me the last echo looked "normal" and was confused when I said, "REALLY??" then laughed and said, "Normal for HIM!" I work with the staff from the cardiology department and I love to tease Warren, one of our echo techs about the time he recognised me but couldn't make the connection to the conversation we'd had the day before over a bed in PICU. His wife is a nurse on another unit and a friend, so she got the whole story.
Here's a related question: What surgical challenge is most significant to the arterial switch patient in the first 24-48 hours post-op? (Sorry Steve, hope your toes are okay!)
0Mar 10, '10 by SteveNNP, MSN, NPVery good answers so far!
Next question: What is the only neonatal GI obstructive disease that does NOT present with bilious vomiting in the neonatal period?
0Mar 10, '10 by Humbled_NurseUsually the proximal obstructions present with bilious vomiting, but the distal obstructions such as hirschsprungs, meconium ileus, and meconium plug syndrome do not present with bilious vomiting. Am I on the right track?
0Mar 10, '10 by ittybabyRNQuote from Humbled_NurseI was also thinking hirschsprungs...Usually the proximal obstructions present with bilious vomiting, but the distal obstructions such as hirschsprungs, meconium ileus, and meconium plug syndrome do not present with bilious vomiting. Am I on the right track?
0Mar 10, '10 by LoveANurse09I thought Hirschsprungs did present with vomiting. Maybe Intussecption(sp?)?I need to research more..
0Mar 11, '10 by littleneoRNWell, I was thinking pyloric stenosis. Because often these kids aren't vomiting until a couple to several weeks of life, when they start with the projectile vomiting. (not usually bilious?) So...it doesn't present with vomiting early in the newborn period, but I suppose technically it is still considered neonatal in the first 30 days. I'm just a little hung up on the exact meaning of the wording of your question.
0Mar 17, '10 by SteveNNP, MSN, NPI'm still here! Sorry... been working a lot lately...haha
The answer is pyloric stenosis. Every other intestinal obstruction whether proximal or distal results in some sort of bilious emesis/aspirates.
Pyloric stenosis usually develops between 2-4 weeks of age.
So let's talk about Hirschprung's disease...
1. How does it present?
2. What neonatal condition is Hirschprung's often associated?
3. What diagnostic tests would you run if you suspected Hirschprung's?
4. Describe briefly the pre and postop nursing care of an infant with Hirschprung's?
0Mar 24, '10 by darynashHirschprung's disease usually presents with failure to pass meconium within 24-48 hours of delivery. Imperforate anus may be associated with Hirschprung's disease. Diagnostically, I would expect a barium enema followed by a rectal biopsy. Preoperatively, I would expect the baby to be NPO and on IV fluids. Also, I would expect enemas to facilitate bowel movements. Postoperatively, I would provide pain management and the baby will be NPO and receiving parenteral nutrition.