Thank you so much for your explanation. I think I kinda get the concept now except the CAD part.
Here’s the part I understood.
the filling pressure is the end-of diastole pressure in the ventricle, referred as preload. And this filling pressure is equal to the pulmonary artery pressure. So, if there is increase in the filling pressure, pulmonary artery pressure increases as well.
Mitral stenosis inhibits complete emptying of left atrium, so the pressure builds up in the lungs. Mitral regurgitation increases filling pressure because it has to accept a higher reload at diastole.
I’m still shaky on the concept of CAD relationship with the filling pressure.
So for the CAD, since the highest peak point of filling pressure is reached faster than in a healthy heart, it causes the pulmonary hypertension (this is what I understood so far).
But, in fact, CAD’s actual filling pressure is lower than in a healthy heart, right? Because if the curve is shifted to the left, the highest point comes first, which is at a lower number in the horizontal axis. It’s just reaching the highest peak filling pressure sooner, causing the pulmonary hypertension more easily? So when you say, “how CAD will give you higher filling pressure,” does this mean it’s just reaching the filling pressure in a much faster rate?
Once again, thank you so much for helping me out…. I’m really feeling hopeless with myself not understanding this…