Published Mar 27, 2014
Nurse_Sian84
9 Posts
Hi Everyone,
I am new here, so please be gentle. So below me I have a case scenario, my assessment is to pick the highest priority out of 5 clinical problems. At first I was going to choose Problem #4 altered blood glucose level .
The reason , because dehydration will take a few hours to rectify, he needs more urgent treatment because he has already got paralysis, his altered BGL is the reason for all the problems listed. I feel that once that is addressed then all other problems will be like a domino effect.
As you can see from below I have changed my intervention to addressing dehydration instead. I had queried this with other uni students and most seem to think that dehydration is it!!
They said I was diagnosing the fact that he may have HHNS when he may be on his way to that state but isnt yet.
The question is to justify my reasoning of why dehydration is of the highest priority, I guess I'm just reaching out for a second opinion, after all if I can justify with a decent rationale then I'm sweet.
Cheers Sian
CASE SCENARIO
Mr George Brown, a 45-year-old male, has been admitted to a ward and has undergone a limb revascularization procedure following experiencing severe leg pain on his anterior right lower leg. The following data were obtained on admission: Subjective data: (reported by Mr Brown's friend) - Diagnosed with type 2 diabetes mellitus 2 years ago - Diagnosed with peripheral arterial disease (PAD) 1 year ago - 2 year history of hypertension - 10 year history of smoking - Taking oral hypoglycaemic medications and anti-hypertensive medications Reported by Mr Brown: - Feeling 'weak' with nausea and vomiting 2 days prior to admission (don't know what has caused him to feel like this) - Extremely thirsty (caused by the dehydration which in turn caused by vomiting and also hyperglycaemia would be aggravating his fluid loss)
- Foot and leg ache continually -( better at rest) (Dehydration causing hypotension which decreases blood flow to the periphery, aggravating the effects of his PAD) Objective data: - Drowsy but can be roused (dehydration) - Dry mouth (dehydration) - Skin dry and warm with decreased turgor (dehydration) - Weight 132 kgs - Height 170 cms - Blood glucose level 21mmol/L (will be increased due to reduced volume of water in his blood - hence increased concentration of all elements in the blood) - Serum potassium level 6mmol/L (as above) normal range 3.5-5.0 mmol/l - Lipid profile: total 253 mg/dl; LDL 91 mg/dl; HDL 25 mg/dl; triglycerides 423 mg/dl (as above) - Diminished peripheral pulses (aggravated with hypotension which accompanies severe dehydration) - Ankle-Brachial Index 0.4 (severe PAD) normal 0.90-1.30, decreased peripheral blood flow due to severe hypotension in a patient with PAD. Previous hospital admission for: - Hyperosmolar Hyperglycaemic Nonketotic Syndrome (State) 2 months ago (>34mmol/l) - ST-segment elevation myocardial infarction 12 months ago Social: - has 3 children visiting, who live with ex-wife - has been working as a real estate agent 20hrs/wk ASSESSMENT Mr Brown presents with multiple clinical problems. Five of Mr Brown's problems are: 1. Altered serum potassium level 2. Dehydration 3. Decreased level of consciousness 4. Altered blood glucose level 5. Paraesthesia (abnormal feelings i.e. pins and needles) and paralysis (loss of ability to move one or all limbs)
bribreeze87
19 Posts
Just wondering is the glucose a typo? 21? Anyways priority wise i would say potassium is the priority. Think about what is gonna kill him first?! His past history of MI and the potential problems related to hyperkalemia?
Hi bribreeze87,
Cheers for your response. Its not a typo the case study given states 21mmol/L, so he is elevated. The potassium level is only slightly raised where as if his insulin levels were >34mmol/L then you would class that as possible HHNS. This is where it gets confusing. Most signs and symptoms of dehydration are also similar of HHNS. As soon as I mentioned dehydration may not be first priority I was shut down immediately.
Hi bribreeze87 Cheers for your response. Its not a typo the case study given states 21mmol/L, so he is elevated. The potassium level is only slightly raised where as if his insulin levels were >34mmol/L then you would class that as possible HHNS. This is where it gets confusing. Most signs and symptoms of dehydration are also similar of HHNS. As soon as I mentioned dehydration may not be first priority I was shut down immediately.[/quote'] were you shut down by your professor or fellow students?
schnookimz
983 Posts
His potassium is more than just "slightly" raised.
were you shut down by your professor or fellow students?
fellow students. I'm still awaiting a response from my tutor (australian term)
Hi Schoonookimz,
thank you for your input, according to Lewis's Medical Surgical Nursing - Assessment and Management of clinical problems it states on page 357 that the normal level is 3.5-5.0mmol/l I guess when you do look at it like that it is elevated...I take back 'slightly'
Esme12, ASN, BSN, RN
20,908 Posts
First an easy way to make the conversion from mg/dl to mmol/L is to divide by 18. To convert mmol/L to mg/dl, multiply by 18.
The glucose is 380.
CASE SCENARIO Mr George Brown, a 45-year-old male, has been admitted to a ward and has undergone a limb revascularization procedure following experiencing severe leg pain on his anterior right lower leg. The following data were obtained on admission: Subjective data: (reported by Mr Brown’s friend) - Diagnosed with type 2 diabetes mellitus 2 years ago - Diagnosed with peripheral arterial disease (PAD) 1 year ago - 2 year history of hypertension - 10 year history of smoking - Taking oral hypoglycaemic medications and anti-hypertensive medications Reported by Mr Brown: - Feeling ‘weak’ with nausea and vomiting 2 days prior to admission (don’t know what has caused him to feel like this) - Extremely thirsty (caused by the dehydration which in turn caused by vomiting and also hyperglycaemia would be aggravating his fluid loss) Could this be from his elevated glucose from his arterial occlusion causing stress aggravated by the dehydration caused by his elevated glucose?Foot and leg ache continually –( better at rest) (Dehydration causing hypotension which decreases blood flow to the periphery, aggravating the effects of his PAD) Could this be caused by the arterial occlusion from the PAD?Objective data: - Drowsy but can be roused (dehydration) - Dry mouth (dehydration) - Skin dry and warm with decreased turgor (dehydration) - Weight 132 kgs - Height 170 cms - Blood glucose level 21mmol/L (will be increased due to reduced volume of water in his blood – hence increased concentration of all elements in the blood) Could this be from his diabeties that is poorly controlled?- Serum potassium level 6mmol/L (as above) normal range 3.5-5.0 mmol/l - Lipid profile: total 253 mg/dl; LDL 91 mg/dl; HDL 25 mg/dl; triglycerides 423 mg/dl (as above) - Diminished peripheral pulses (aggravated with hypotension which accompanies severe dehydration) and severe PAD- Ankle-Brachial Index 0.4 (severe PAD) normal 0.90-1.30, decreased peripheral blood flow due to severe hypotension in a patient with PAD. Previous hospital admission for: - Hyperosmolar Hyperglycaemic Nonketotic Syndrome (State) 2 months ago (>34mmol/l) - ST-segment elevation myocardial infarction 12 months ago Social: - has 3 children visiting, who live with ex-wife - has been working as a real estate agent 20hrs/wk ASSESSMENT Mr Brown presents with multiple clinical problems. Five of Mr Brown’s problems are: 1. Altered serum potassium level 2. Dehydration 3. Decreased level of consciousness 4. Altered blood glucose level 5. Paraesthesia (abnormal feelings i.e. pins and needles) and paralysis (loss of ability to move one or all limbs)
Mr George Brown, a 45-year-old male, has been admitted to a ward and has undergone a limb revascularization procedure following experiencing severe leg pain on his anterior right lower leg. The following data were obtained on admission: Subjective data: (reported by Mr Brown’s friend) - Diagnosed with type 2 diabetes mellitus 2 years ago - Diagnosed with peripheral arterial disease (PAD) 1 year ago - 2 year history of hypertension - 10 year history of smoking - Taking oral hypoglycaemic medications and anti-hypertensive medications Reported by Mr Brown: -
Feeling ‘weak’ with nausea and vomiting 2 days prior to admission (don’t know what has caused him to feel like this) - Extremely thirsty (caused by the dehydration which in turn caused by vomiting and also hyperglycaemia would be aggravating his fluid loss) Could this be from his elevated glucose from his arterial occlusion causing stress aggravated by the dehydration caused by his elevated glucose?
Foot and leg ache continually –( better at rest) (Dehydration causing hypotension which decreases blood flow to the periphery, aggravating the effects of his PAD) Could this be caused by the arterial occlusion from the PAD?
Objective data: - Drowsy but can be roused (dehydration) - Dry mouth (dehydration) - Skin dry and warm with decreased turgor (dehydration) - Weight 132 kgs - Height 170 cms - Blood glucose level 21mmol/L (will be increased due to reduced volume of water in his blood – hence increased concentration of all elements in the blood) Could this be from his diabeties that is poorly controlled?- Serum potassium level 6mmol/L (as above) normal range 3.5-5.0 mmol/l - Lipid profile: total 253 mg/dl; LDL 91 mg/dl; HDL 25 mg/dl; triglycerides 423 mg/dl (as above) - Diminished peripheral pulses (aggravated with hypotension which accompanies severe dehydration) and severe PAD- Ankle-Brachial Index 0.4 (severe PAD) normal 0.90-1.30, decreased peripheral blood flow due to severe hypotension in a patient with PAD.
Previous hospital admission for: - Hyperosmolar Hyperglycaemic Nonketotic Syndrome (State) 2 months ago (>34mmol/l) - ST-segment elevation myocardial infarction 12 months ago
Social: - has 3 children visiting, who live with ex-wife - has been working as a real estate agent 20hrs/wk ASSESSMENT Mr Brown presents with multiple clinical problems.
Five of Mr Brown’s problems are: 1. Altered serum potassium level 2. Dehydration 3. Decreased level of consciousness 4. Altered blood glucose level 5. Paraesthesia (abnormal feelings i.e. pins and needles) and paralysis (loss of ability to move one or all limbs)
Mr George Brown, a 45-year-old male, has been admitted to a ward and has undergone a limb revascularization procedure following experiencing severe leg pain on his anterior right lower leg. The following data were obtained on admission:
Subjective data:
reported by Mr Brown’s friend - Diagnosed with type 2 diabetes mellitus 2 years ago - Diagnosed with peripheral arterial disease (PAD) 1 year ago - 2 year history of hypertension - 10 year history of smoking - Taking oral hypoglycaemic medications and anti-hypertensive medications
Reported by Mr Brown: - Feeling ‘weak’ with nausea and vomiting 2 days prior to admission - Extremely thirsty Foot and leg ache continually –( better at rest)
Objective data: - Drowsy but can be roused - Dry mouth - Skin dry and warm with decreased turgor - Weight 132 kgs - Height 170 cms - Blood glucose level 21mmol/L - Serum potassium level 6mmol/L normal range 3.5-5.0 mmol/l - Lipid profile: total 253 mg/dl; LDL 91 mg/dl; HDL 25 mg/dl; triglycerides 423 mg/dl - Diminished peripheral pulses - Ankle-Brachial Index 0.4 (severe PAD) normal 0.90-1.30,
. Previous hospital admission for: - Hyperosmolar Hyperglycaemic Nonketotic Syndrome (State) 2 months ago (>34mmol/l) - ST-segment elevation myocardial infarction 12 months ago Social: - has 3 children visiting, who live with ex-wife - has been working as a real estate agent 20hrs/wk
ASSESSMENT Mr Brown presents with multiple clinical problems. Five of Mr Brown’s problems are: 1. Altered serum potassium level 2. Dehydration 3. Decreased level of consciousness 4. Altered blood glucose level 5. Paraesthesia (abnormal feelings i.e. pins and needles) and paralysis (loss of ability to move one or all limbs)
Mr Brown presents with multiple clinical problems. Five of Mr Brown’s problems are:
1. Altered serum potassium level
2. Dehydration
3. Decreased level of consciousness
4. Altered blood glucose level
5. Paraesthesia (abnormal feelings i.e. pins and needles) and paralysis (loss of ability to move one or all limbs)
You prioritize according to what is going to kill him first. So looking here what is most important?
Potassium. An elevated K can cause lethal cardiac arrythmias. Why is this patients glucose elevated? How does and elevated glucose impact the elevated K? http://www.elsevierhealth.com/media/us/samplechapters/9780443071973/9780443071973.pdf
The next would be dehydration....why is he dehydrated? Well he stated he had nausea and vomiting prior to admission. Why was he vomiting? Well....he has a history of HHNK. The chances this has reoccurred are high. Is his elevated sugar causing nausea and vomiting due to the stress his body being stressed from an occlusion of his lower leg? I think his compliance might be an issue as well.
Altered blood glucose. The elevated glucose will affect healing and contributes to his overall health picture.
Decreased LOC. Patients with elevated glucose that are dehydrates with nausea and vomiting and dehydration will have a decreased LOC and an elevated K
Now looking at these symptoms what disease process causes these symptoms? the hint is in his PMH
Pain. Your patient has pain.
He is at risk for further peripheral circulatory occlusions and infection due to his disease process, poorly maintained health (still smoking), and fresh post op
What are your thoughts? Do you use NANDA I? What semester are you? What did your instructor say?
Yeah what did you professor (tutor) say? I wanted to think about the potassium and how it connects to the patient and what could potentially happen? In prioritization always think what will
I tried to edit my reply and it sent
It unfinished! Sorry bout that. Let me know :-)
l1234567
67 Posts
Hey I'm doing this case study too!
It's dehydration from what I've found out so far and that is what my tutor said as well. From starting him on a sodium chloride infusion it will prompt diuresis and also the blood level will be higher so the ratio to potassium will be lower. Also you need to start a sodium chloride infusion as this is done before a attacking his bsl (journals). His circulation will also be improved and loc. His paralyiss will be improved as well.
I have a question too!
the second question of the case study is
"Mr Brown has a further problem of an orally taken temperature of 39o C. He is complaining of general
discomfort related to his body temperature. Identify the best management for the above problem in
Mr Brown’s case. Using an academic writing style, justify your decision for Mr Brown’s body
temperature management in this admission with a synthesis of evidence-based literature. (approx.
750 words). "
Apparently he has an infection and we have to find out what sort of infection it is (via my tutor) is osteomylitis a good one? why/ why not?
Where is this paralysis? Paresthesias are NOT paralysis.
Paresthesia is a sensation of tingling, tickling, prickling, pricking, or burning of a person's skin with no apparent long-term physical effect. The manifestation of a paresthesia may be transient or chronic. The most familiar kind of paresthesia is the sensation known as "pins and needles" or of a limb "falling asleep".
Paralysis is loss of muscle function for one or more muscles. Paralysis can be accompanied by a loss of feeling (sensory loss) in the affected area if there is sensory damage as well as motor. Paralysis is most often caused by damage in the nervous system, especially the spinal cord or brain.
Mr Brown has a further problem of an orally taken temperature of 39o C. He is complaining of general discomfort related to his body temperature. Identify the best management for the above problem in Mr Brown’s case. Using an academic writing style, justify your decision for Mr Brown’s body temperature management in this admission with a synthesis of evidence-based literature. (approx. 750 words). "