Published Nov 10, 2013
Tmari1
12 Posts
Okay..
My patient was shot in the head behind the left ear, suffering a traumatic brain injury. Subsequently, patient suffered subarachnoid hemorrhage, subdural hematoma, edema, and (+) midline shift. Patient has since developed tracheal erosion r/t tracheostomy placement and c-difficile infection most likely r/t prophylactic broad spectrum antibiotics.
I am doing a long careplan on this patient and am almost finished except for one more detail...this patient has hyperphosphatemia and I can not figure out why. We have to analyze our labs in our careplan. My patient does not have any renal issues, hasn't had any blood transfusions in 3 weeks, and has no muscle necrosis, so why in the world would she have an increased phosphorous level?? Could anybody help please because I am just stumped?
The only reason I can think of is from tissue damage from her brain injury or from the tracheal erosion... but neither of those are muscle tissue, persay, so I don't feel like it's a strong reason for her to have hyperphosphatemia. Also, her calcium is not low.
I'm just stumped.
Can anybody help me please?
DatMurse
792 Posts
whats his HCT?
Renal function?
Severe hyperphosphatemia associated with hemo... [Am J Emerg Med. 1992] - PubMed - NCBI
She has hct 19.6 and hgb 9.5. However, she has no impairment in her renal function. She has adequate voiding/uop and her BUN and CR levels are WNL.
what is the phosphate at and is there anything on the GFR? When was she admitted and how long has she been hyperphosphatemia?
what medications is she on? is anything causing retention of phosphate?
Admitted 3 weeks ago. Phosphate 4.9- high for 4 days. She's on nexium, dilaudid, propranolol, heparin, and flagyl
deficiency in mag or calcium?
http://www.biomedcentral.com/1471-2369/14/178
New study with connection to anemia
Respiratory acidosis is another factor if they have that.
Esme12, ASN, BSN, RN
20,908 Posts
YOu patient has HUGE reasons for hyperphosphatemia.......like the gunshot wound, immobility....have they received tube feedings or TPN?
Patients with hyperphosphatemia most commonly complain of muscle cramping secondary to low calcium levels. This may progress to tetany, delirium, and seizures. A search for the following historical clues may help identify those patients at risk for increased phosphorus levels. Renal diseasePast or present hemodialysis Adherence to renal (low phosphorus) diet Use of oral phosphate binders [*]CancerLeukemia Lymphoma Bone tumors Other cancers Chemotherapy treatment [*]Endocrinopathies [*]Trauma [*]Burns or heat-related illnesses [*]Prolonged immobilization [*]Metabolic or hematologic disorders including genetic predisposition [*]MedicationsOral phosphate binders Potassium phosphate Antacid use Bisphosphonate therapy [*]Use of laxatives (oral/rectal) and enemas [*]Use of nutritional supplements or hyperalimentation [*]Ischemic bowel (possible phosphorus elevation)
[*]Cancer
[*]Endocrinopathies
[*]Trauma
[*]Burns or heat-related illnesses
[*]Prolonged immobilization
[*]Metabolic or hematologic disorders including genetic predisposition
[*]Medications
[*]Use of laxatives (oral/rectal) and enemas
[*]Use of nutritional supplements or hyperalimentation
[*]Ischemic bowel (possible phosphorus elevation)
The most common cause of hyperphosphatemia is renal failure. Less common causes can be classified according to pathogenesis, ie, increased intake, decreased output, or shift from the intracellular to the extracellular space. Often, several mechanisms contribute. Impaired renal excretion is most frequently the major factor, with relatively increased intake or cell breakdown contributing to the problem. http://emedicine.medscape.com/article/241185-clinical#a0218
Increased intakeExcessive oral or rectal use of an oral saline laxative (Phospho-soda) Excessive parenteral administration of phosphate Milk-alkali syndrome Vitamin D intoxication [*]Decreased excretionRenal failure, acute or chronic Hypoparathyroidism Pseudohypoparathyroidism Severe hypomagnesemia Tumoral calcinosis Bisphosphonate therapy [*]Shift of phosphate from intracellular to extracellular spaceRhabdomyolysis Tumor lysis Acute hemolysis Acute metabolic or respiratory acidosis [*]SpuriousBlood sample taken from line containing heparin or alteplase[28, 29] High concentrations of paraproteins[30] Hyperbilirubinemia[31] In vitro hemolysis Hyperlipidemia[32]
[*]Decreased excretion
[*]Shift of phosphate from intracellular to extracellular space
[*]Spurious
Thank you so much!!!!! I've been using Nursing Databases... guess I should have expanded my search to google and so on.. haha Thank you!!!
You're welcome....:)
yea.. Dont do what I did.
I looked for a jacket everywhere but the closet
taydian
33 Posts
Holy crap. I really admire medical nurses. I'm not demeaning psych at all by any means......... but medical nursing. Much love and respect to you all.