so i have this case study and i'm kind of unsure of how this is all related i put some answers but would just like some feedback plese :)
[color=#c3260c]
[color=#c3260c]*[color=#404040]t.c. is an 80 year-old farmer who is diabetic. his history includes smoking for 50 years (but not in the past 10 years), angina, hypertension, and atrial fibrillation. t.c. has been on nifedipine (procardia) 20 mg qid[color=#404040]and digoxin (lanoxin) 0.375 mg qd. he adjusts his insulin (regular and nph) depending on his activity (he occasionally helps his sons with livestock and field work). t.c. underwent triple coronary bypass surgery yesterday.
[color=#404040]the postoperative course was uncomplicated until it was determined in the postanesthesia[color=#404040]reco[color=#404040]veryareathat he was bleeding. t.c. was returned to surgery and five units of blood were administered during the second operation. today, t.c.'s urine output is less than 5 ml/hr and he is diagnosed with acute tubular necrosis (atn).[color=#404040]
since t.c.'s blood pressure never dropped below 80/50 in the recovery area and surgery, what contributed to the poor kidney perfusion that led to acute tubular necrosis? consider his original medical problems.
these are my answers any feedback would be greatly appreciated!!!!
so i have this case study and i'm kind of unsure of how this is all related i put some answers but would just like some feedback plese :)
[color=#c3260c]
[color=#c3260c]*[color=#404040]t.c. is an 80 year-old farmer who is diabetic. his history includes smoking for 50 years (but not in the past 10 years), angina, hypertension, and atrial fibrillation. t.c. has been on nifedipine (procardia) 20 mg qid[color=#404040] and digoxin (lanoxin) 0.375 mg qd. he adjusts his insulin (regular and nph) depending on his activity (he occasionally helps his sons with livestock and field work). t.c. underwent triple coronary bypass surgery yesterday.
[color=#404040]the postoperative course was uncomplicated until it was determined in the postanesthesia[color=#404040]reco[color=#404040]very area that he was bleeding. t.c. was returned to surgery and five units of blood were administered during the second operation. today, t.c.'s urine output is less than 5 ml/hr and he is diagnosed with acute tubular necrosis (atn).[color=#404040]
since t.c.'s blood pressure never dropped below 80/50 in the recovery area and surgery, what contributed to the poor kidney perfusion that led to acute tubular necrosis? consider his original medical problems.
these are my answers any feedback would be greatly appreciated!!!!
cvd(cardiovascular disease), htn (increase pressure