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Hi, I'm trying to work on my concept map & I'm just not sure if I'm doing it correctly or not...Our program gave us like a 5 minute lecture on these maps & everyone seems to be lost on it....Anyways here goes....
My diagnosis was: Hyponatremia
Reason for needing healthcare: Falls
Here's some key problems NANDA Diagnoses & supporting data I have so far:
- Altered thought processes r/t neurological dysfunction
1.) Progressive dementia
2.) 65+
3.) Progressive dementia
4.) Reminding patient to watch fluid intake
5.) CT Head results
- Risk for falls r/t diminished mental status
1.) 65+
2.) Patient requires use of assistive devices
3.) History of falls
4.) CT head results
5.) PT results; staggering right leg possibly r/t CT results.
- Fluid volume excess r/t increase in ADH & H2O retention
1.) Low serum Na
2.) SIADH
3.) Water intoxication, no signs of edema, pt on
4.) Patient complaining of headache & nausea.
- Excess fluid volume r/t excessive intake of hypotonic fluids
1.) Patients poor appetite but drinks a lot of fluids.
2.) I wanna say intake > output; however, my patient's output was higher than their intake but they did have a high PVR earlier in the day like of 300 mL.
I'm connecting a lot of these boxes together with one another & just see 3 more key problems. But just curious, does that look like I'm doing this right?
Ok so you have hyponatremia....Hyponatremia is defined as a serum sodium level of less than 135 mEq/L and is considered severe when the serum level is below 125 mEq/L. Many medical illnesses, such as congestive heart failure, liver failure, renal failure, or pneumonia, may be associated with hyponatremia. "Mild" hyponatremia is defined as a serum sodium concentration between 130 and 135 mmol/L, "moderate" hyponatremia as a concentration between 125 and 129 mmol/L, and "profound" hyponatremia as a concentration less than 125 mmol/L. Symptoms range from nausea and malaise, with mild reduction in the serum sodium, to lethargy, decreased level of consciousness, headache, and (if severe) seizures and coma. Overt neurologic symptoms most often are due to very low serum sodium levels (usually
PVR = Post Void Residual.
Or basically the amount of urine left in the bladder after urination. When we did the bladder scan at about 10 AM she put out about 175 mL but had 300 mL left over. Then at 1240 PM she had an output of 535 mL with a PVR of about 100mL left over.
By this point in time she had an input of 575 mL.
NANDA I describs excess fluid volume as: Increased isotonic fluid retention
Defining Characteristics (which your patient must exhibit one or more of these symptoms or evidence)
Adventitious breath sounds; altered electrolytes; anasarca, anxiety, azotemia, blood pressure changes; change in mental status; changes in respiratory pattern, decreased hematocrit, decreased hemoglobin, dyspnea, edema, increased central venous pressure; intake exceeds output, jugular vein distention, oliguria; orthopnea; pleural effusion; positive hepatojugular reflex; pulmonary artery pressures; increased pulmonary congestion; restlessness; specific gravity changes; S3 heart sound; weight gain.
Related Factors (r/t): as evidenced by
Compromised regulatory mechanism; excess fluid intake; excess sodium intake.
To say that she has SIADH it needs to be proven and documented. Now why is she drinking so much? Is her glucose elevated? Is this a presentation of dementia?
OK...to an ICU nurse PVR is pulmonary vascular resistance. Something altogether different....LOLPVR = Post Void Residual.Or basically the amount of urine left in the bladder after urination. When we did the bladder scan at about 10 AM she put out about 175 mL but had 300 mL left over. Then at 1240 PM she had an output of 535 mL with a PVR of about 100mL left over.
By this point in time she had an input of 575 mL.
OK now you have another problem...Urinary Retention
NANDA describes Urinary Retention as Incomplete emptying of the bladder
[h=4]Defining Characteristics[/h] Absence of urine output; bladder distention; dribbling, dysuria; frequent voiding; overflow incontinence; residual urine; sensation of bladder fullness; small voiding
[h=4]Related Factors (r/t)[/h] Blockage, high urethral pressure, inhibition of reflex arc, strong sphincter
Is she diabetic?
SIADH is a medical condition The syndrome of inappropriate antidiuretic hormone (ADH) secretion (SIADH) is defined by the hyponatremia and hypo-osmolality resulting from inappropriate, continued secretion or action of the hormone despite normal or increased plasma volume, which results in impaired water excretion.I've already got my pathophysiology paper done on it too.When researching on it, I assumed my patient had euvolemic hyponatremia which is related to SIADH & can be caused by dilutional hyponatremia, if that makes sense.
She may have low Na due to euvolemic hyponatremia...but this can also be caused by excessive water intake....or water intoxication. Water intoxication, also known as water poisoning or dilutional hyponatremia, is a potentially fatal disturbance in brain functions that results when the normal balance of electrolytes in the body is pushed outside safe limits by over-hydration.
In nursing never ASSume.Thanks for all the help, this is really helping me out.And her glucose was elevated, which I didn't really think of that. And no she isn't diabetic. So I wasn't sure why the glucose was elevated, I assumed labs were drawn after a meal.
What are the symptoms of hyperglycemia? Even learned MD"s cam miss the simplest of things. If she has gone a long time without diagnosis this might explain why she doesn't empty her bladder.
The three biggies...
Ok.....so now I see many other diagnosis and potential problems for your patient.
Nausea: Actual you said she was nauseated
Acute Pain she has a headache...which can be from cerebral edema from water intoxication and low Na
Impaired physical Mobility: she is having difficulty walking with weakness and a hx of falls.
Risk for Injury: this is a high risk as she has already fallen in the past
Risk for unstable blood Glucose level: until you determine if their is an issue is is a concern.
Risk for Falls: high risk for present because she has fallen in the past and has an unsteady gait.
Acute Confusion: unknown at this time
Chronic Confusion: no real history but has been mentioned.
All these have their own definitions and characteristics/symptoms in NANDA I. They are in your Ackley book I use it too. Do you have the 10th edition?
do you see how I am getting these?
Look at those sheets I posted a a couple of posts ago....they will help you organize your thinking.
K I do, & will do! Thank you a bunch, this is helping out a ton. As for my etiologic factors, they'll always be my related to something. So, like if I pick Impaired physical mobility it be: Impaired physical mobility related to neurological impairment.
For supporting evidence do you think I could put something such as....Possibly not r/t low sodium serum because as levels increase symptoms don't improve?
Little_Bear2013
105 Posts
Ya, sorry I should've mentioned that. On admission, Na levels were like 124 on 3/4/14 but when we did clinicals last Friday on 3/7/14 they were at 131, so they are improving but they are still a little low. Hematocrit was also pretty low, at about 33.4% which the reason why I thought it was low was because of increased intravascular volume due to dilution.
Also, her hgb was low at about 11.3 which I thought may be due to her poor diet which causes a lack of folic acid & vitamin B12.
Probably should have mentioned lab values too, duh.