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where are you going to school? i am from illinos too.i hope this will help:
alpha 1 and beta 2 blockers: doxazosin, prazosin, terazosin; block alpha adrenergic receptors in ANS;
S yncope, sexual dysfunction
I increase drowsiness, , HR, othrostatic hypotension
N eed to recline for 3-4 hours after first dose
beta 1 blockers: decrease stimulation to heart;
acebutolol, atenolol, esmolol, metoprolol;
beta 2 blockers: constrict airways and uterus;
Bradycardia
Libido decreased, lipids increased
brOnchospasm
Chf, and conduction abnormalities
Known hypotension
Exhaustion, emotional depression
Reduced recognition of hypoglycemia
ACE INHIBITORS: lisinopril, captopril, enalapril
Action:prevent conversion of angiotensin I to II
Pruritis
Rash
Incresed hypotension, tachycardia, angioedema, cough, infection
Lously taste (for about 3 months) and headache
CALCIUM CHANNEL BLOCKER
verapamil, diltiazem, nifedipine
Action: block calcium access to cell
Hypotension, headache
Edema
Constipation
Know to watch for worsening of CHF and heart block
that are tips from my pharm teacher which i found helpfull;
good luck to you, :)
no tips for diuretics, just need to study those. i got only one cumulative:
Diet - low sodium intake and increase potassium (not for potassium sparing diuretics this one)
Intake and output monitor for edema and CHF complication
Undesirable effects: fluid and electrolytes imbalance, dizziness, hypotension
Reasses BP, HR, and electrolytes
Elderly: have dicreased liver and kidney function, monitor for toxicity, electrolytes imbalance
Take am and if bid before 6pm to prevent nocturia
Increase risk for orthostatic hypotension, teach pt to rise slowly
Cancel alcohol input
hope it helps, good luck, mine exam on this is following monday too, :)
Renin-angiotensin system is a baroreflex mechanism that controls arterial pressure. It is a powerful mechanism for controlling pressure by the kidney.
Renin is released by the kidneys when the arterial pressure falls to low. Most of the renin enters the renal blood and passes out of the kidneys to circulate throughout the entire body. However, small portions of it remain in the kidneys and initiate several intrarenal functions one of which is the release of angiotensin I. Angiotensin I is a mild vasoconstrictor and has no significant change in the circulatory function. Angiotensin I is coverted by Angiotensin-Coverting Enzyme ( Peptidyl Dipeptidase ) into Angiotensin II which is a very potent vasoconstrictor. Angiotensin II's vasoconstricting effects are:
1. constrict the arterioles increasing pressure
2. decrease the excretion of salt and water. As salt and water accumulates, there is an increase in ECF volume thereby increasing pressure.
So, with this mechanism in mind, ACE inhibitors blocks the conversion of angiotensin I to angio II. Aside from this, ACE inhibitors release bradykinin which is a potent vasodilator.
Renal physiology is a very hard concept but once you understand it, you are going to be fine.
xokelly2
150 Posts
I am having a hard time differentiating between some of the most common drug classes R/T cardiac: ACE Inhibitors, beta-blockers, and Calcium channel blockers. Does anyone have any special memerization techniques that helps you remember the important stuff??? Thanks xokell