Originally posted by jobear
Matt, I am wondering about the ejection fraction and alterations in blood pressure related to hypertrophy of the hearts wall. If the kidneys and other hormone responses are working correctly and the blood pressure is low the SG will be increased due to retention of salt and water to maintain blood pressure thus making the problem worse! If laxis is taken to pull water off the lungs, then SG will be low due to diuresis. What do you think?
Yes, Steph, you are correct that once the renin-angiotensin-aldosterone system has kicked in, it will make matters worse and the SG will be higher. Remember, though, that the body has several compensatory mechanisms to deal with decreased blood flow. The first is increase of heart rate, the second is increasing the peripheral vasoconstriction. While these mechanisms are working, the SG will be normal.
Personally, I have never measured the SG of a patient in CHF. I don't see how it would be relevant -- what information would it give you that you need for treatment? If this is a question on a test, then it is for academic purposes only, not clinical.