To follow up what CRNA, DNSc and crna29 have said, but in light that you have already written some of the paper-------let me see if this helps any.
Anesthesia for open heart surgery in the past, was often done primarily with high dose narcotics. This technique is very cardiovascular stable-meaning it does not depress the myocardium in its pumping ability.
The down side, is all those narcotics take time to wear off, and patients were ventilated for days. This resulted in more effort to individualize the anesthetic. A patient with a strong heart, and preserved LV function does not need a high dose narcotic technique. This patient might have an anesthetic not too different from that for any other surgery, including the use of inhalation agents (which are known for their cardiovascular depression).
On the other hand, if there is already myocardial damage, too much inhalation agent can be detrimental, by further weakening an already weakened pump.
(This may be more than you wanted to know, but these are not simple questions to discuss). So, one angle you might try is the inhalation agent vs. narcotic decision.
Another angle could be based on the fact that the only thing that makes a cardiac anesthetic different from that for any other surgery, is the use of the cardiac pulmonary bypass pump. (Which is part of the reason you couldn't find anything by looking up "cardiac anesthetic agents"). You could talk about the pump, but there are really only a few pharmacology issues. Doses are diluted out by the increased volume of the pump, need to (or not) redose when you begin to warm the patient (accompanied by an increase in metabolism). The need for heparinazation, and its reversal at the end of surgery. A big issue in cardiac anesthesia is supporting the patient as they come off pump, but the drugs in question then are not "anesthetic" in nature. For that we are talking about volume replacement, transfusion, inotropes, vasodilators/constrictors.
Hope this has helped a bit. Difficult subject to narrow down adequately.