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MCCD Spring 2014 - Orientation packets
Thanks for all the information!
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MCCD Spring 2014 - Orientation packets
Where are you purchasing the uniform scrubs? I haven't received my orientation packet... and wont be getting it till around December:sarcastic:
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Human A&P 202 Review: Osmolarity of Body Fluids
From: Human Anatomy & Physiology- Elaine N. Marieb & Katja Hoehn
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Human A&P 202 Review: Blood Glucose Regulation
I used Human Anatomy & Physiology (eight edition)- Elaine N. Marieb & Katja Hoehn (I paraphrased).
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Human A&P 202 Review: Blood Glucose Regulation
Blood Glucose Regulation - Alpha cells are glucagon- synthesizing - Beta cells are insulin-producing They act as fuel sensors secreting glucagon and insulin during fasting and fed states. Glucagon and insulin are intimately but independently involved in the regulation of blood glucose levels. Effects are antagonistic; insulin is hypoglycemic hormone glucagon is a hyperglycemic hormone. Falling blood glucose levels (humoral stimuli) prompt the alpha cells to secrete glucagon. Other stimuli include sympathetic nervous system stimulation and rising amino acid levels. Glucagon targets the liver where it breaks down glycogen to glucose; glucose is then released into the blood by liver cells, causing blood glucose levels to rise. Glucagon release is suppressed by rising blood glucose levels, insulin and somatostain (GHIH). Pancreatic beta cells are stimulated to secrete insulin chiefly by elevated blood glucose levels (also by rising plasma levels of amino acids and fatty acids). Insulin stimulates glycogen production and inhibits the breakdown of glycogen to glucose in the liver. Insulin also stimulates amino acid uptake and protein synthesis in muscle tissue. Insulin sweeps glucose out of the blood causing it to be used for energy or converted to other forms. As body cells take up glucose and other nutrients, and plasma levels of these substances drop, insulin secretion is suppressed.
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Maricopa Waiting List / Placement / Timestamps 2010+
If that is the case that is very very suspicious. Unless Randallk, did not re-affirm before they did placement, that is one way to get passed over.
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Fall 2013 maricopa mursim program accepted date stamps
randallk: You have not been placed? I'm not sure who you were asking, but I don't know anything about any of the programs (sorry for being of no help). Also I just thought I'd let you know that I know of someone who got placed at gateway accelerated and will be deferring. So that's one spot open there.
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Human A&P 202 Review: Osmolarity of Body Fluids
I'm posting Human A&P 202 topics, to help me review for nursing school; hopefully they will be helpful for students currently taking BIO202. Osmolarity of Body Fluids Na+ and K+ regulation are crucial to overall body homeostasis. Their regulation is the primary job of aldosterone (a mineralocorticoid). Changes is Na+ concentration lead to changes in blood volume, blood pressure and rising levels of K+ lead to aldosterone secretion by the adrenal cortex. The regulation of a number of ions including K+, H+, HCO3- and Cl- is coupled to that of Na+. Aldosterone reduces excretion of Na+ from the body. It targets the distal parts of the kidney tubules, where it stimulates Na+ reabsorption and water retention along with the elimination of K+. The renin-angiotensin mechanism influences both blood volume and blood pressure by regulating the release of aldosterone and therefore Na+ and water reabsorption by the kidneys. Specialized cells in the kidneys release renin into the blood when blood volume/pressure declines. Renin initiates a cascade that produces Angiotensin II; an aldosterone stimulator. Fluctuation blood levels of K+ directly influence the zona glomerulosa cells in the adrenal cortex. Increased K+ stimulates aldosterone release. Atrial natriuretic peptide hormone is secreted by the heart when blood pressure rises, fine tunes blood pressure and sodium-water balance of the body. It blocks renin and aldosterone secretion, consequently ANP'S overall influence is to decrease blood pressure by allowing Na+ and water to flow out of the body in urine.
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Fall 2013 maricopa mursim program accepted date stamps
I was placed at EMCC as well :) Are you still moving to Paris?
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Maricopa Waiting List / Placement / Timestamps 2010+
I got placed!!! 6/10/11 8:02:28
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Maricopa Nursing Date Time Stamp Question
I was placed today! Time stamp: 6/10/11 @ 8:02:28 am
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Fall 2013 maricopa mursim program accepted date stamps
I was actually placed today!!!!!!!!!!!!! Time stamp: 6/10/11 @ 8:02:28 am.
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Fall 2013 maricopa mursim program accepted date stamps
RN2b23: Wow!!! That is shocking news, it definitely seems like a cruel turn of events. There must be a special opportunity waiting for you in Paris, there just has to be (change= opportunity).
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Fall 2013 maricopa mursim program accepted date stamps
I just called the nursing hot line, and was told that I will most likely not get in for this spring (I only have one choice). So I guess its a 3+ year wait for me.
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Fall 2013 maricopa mursim program accepted date stamps
Hello Trig Your time stamp is 8:01:33, that's 60 seconds (1 minute) + 33 seconds, so that would make you 93 in your group. Each second is one person on the list. In my opinion 93 is not that bad, I'm 148 in my group, so you could have been worse off. As far as you getting called next year, it's hard to say. There are still people from February (very few), April, June, August and 92 from October still ahead of you. Once they place this Tuesday, I think you will have a better idea of when they might call you. For me the wait has been a little over 2 years so far, If I don't get placed this spring then It will be a 3 year wait for me (if i get placed for fall of 2014, If i don't then it will be a 3 1/2 year wait), so i guess it is possible to wait that long. I have my reasons why I cant go the CEP route, but that is a faster way to get in. Someone from the October 2011 group posted that they either, already started (the nursing program) or are going to start soon (they went the CEP route).