SBURNSTEVEN

My preceptor held cricoid pressure while I intubated a patient considered a full stomach with RSI. After I intubated and inflated the cuff, my preceptor kept holding cricoid pressure and asked "Should I let go?" The answer was no but I didn't know why and can't find it when I try to look it up.....does anyone know why?

I had a patient that came in with Pancreatitis. By day 2, Lipase and Amylase were trending down, and the patient didn't complain of pain. They went into respiratory distress and had a distended abdomen. (large abdomen made it difficult to breath). Abdominal X-ray showed an Ileus. Patient has absent bowel sounds......Was the Ileus narcotic induced (patient had hydromorphone 0.5 mg q3h for 5 doses), or could it be associated with the pancreatitis? Also, the NGT placed to "decompress" I believe is to remove the gas from the intestines and not the gastric contents (acids) which do also come out with the NGT? Patient has been NPO since arrival. The NGT to LCWS had only made his abdomen go down a small amount.....how long until his abdomen goes down or the Ileus resolves?

How does a patient with COPD and therefore chronically Hypercapnia not have a respiratory acidosis (acute decompensation due to respiratory infection can cause respiratory acidosis)....but why not normally? I'm assuming because of their kidneys compensation with bicarbonate?

Can someone pleas help me understand this....I came across this in a CCRN book I'm using as a study guide... For a low-flow oxygen delivery system (assuming nasal cannula); FiO2 is dependent on rate and depth of ventilation and fit of device. If minute ventilation increases, oxygen concentration decreases because the amount of room air (diluent) increases in relation to the amount of oxygen via the oxygen delivery system.

Thanks, I've been working on a med-surg floor for a year and a half and I'm orientating in CT ICU now.

Let's just focus on CVP.....normal is about 1-8 (depending on source). Since the patient is Post CABG is the normal CVP inadequate because we know the patient lost blood? Should we aim for a CVP around 10 as we tut rate down Levophed? This seems to be how it's done on the ct ICU floor I am orientations on.

Thanks

I'm an RN. I know it must be due to an abnormality in the conduction system, but where?

Why are Minimally invasive Aortic valve replacement patients at higher risk for asystole postoperatively? They have their pacing wires intact up to 72 hours post op which is longer than other cardiac surgical procedures.

Post op day 0. H/h was 9.1/27.7 Albumin was given. Please explain why? Wouldn't the Albumin dilute the h/h further? Blood wasn't given, the reasoning was because blood products have a higher incidence of causing lung injury in intubated patients.

When a patient has open heart surgery, they loose an "unobtainable" amount of blood in the OR as per the post op note. The nurse receives this patient right after their procedure and their blood pressure is labile. Why is their blood pressure like this? When the patient was at 170, from going from SBP 70 to 170 back and forth, Albumin was given. I would assume with a SBP of 170 this would be bad, but the blood pressure was not just 170 but up and down. The rationale I heard was because the patient was 'dry'; they lost blood in the OR obviously. Someone help me to understand why/how Albumin would help this patient.