ChrisSmith

I was struggling with applying more basic concepts and eventually found a nice simple explanation here: CV Physiology: Central Venous Pressure Thanks for your reply Belgian which was also very helpfull. I am just a few weeks into my CCU training. They work with 'competentie gericht onderwijs' (competence orientated education??) but here that is applied more on the workfloor than in the classroom.

This is homework in that I am a student nurse (in Holland) and am trying to figure this out... the question I posed myself. I am on holiday at the moment and therefore not at college or at work with people immediately available to ask and am having trouble finding an answer in my books and on the internet.I stumbled upon this site and thought why not post here and see if anyone can help... Does this justify my question??? Most of the information I have found deals with cardiogenic shock where the cardiac output is reduced due to stroke volume reduction as is the case with infarction for example and compensation is with increased frequency. In this case CPV increases though I am going round in circles trying to understand the mechanisms which cause this... And when bradycardia is the cause of reduced cardiac output and we have Adams Stokes syndroom do we also speak of cardiogenic shock?

A patient has extreme bradycardia, 30 bpm, with a 3rd degree AV block. Cardiac output is reduced due to slow frequency. Contractility and TPR are increased as compensation but what happens to CVP and why?