Dsmcmm

The R-R intervals change by 80ms-40ms-80ms. They are not consistent as prior to the PVC. Given the changes are small, but there is change to rhythm. A conduction delay with any type of heart block could keep the HR below 100 and further more below 70. the method I used was to mark the rhythm prior to and after PVC and transfering to R-R. I see no P waves during the episode of (artifact or Afib) leading to my questions. There are many documented cases of (athletic Lone afib) with HR well below 70

The R-R intervals change by 80ms-40ms-80ms. They are not consistent as prior to the PVC. Given the changes are small, but there is change to rhythm. A conduction delay with any type of heart block could keep the HR below 100 and further more below 70. the method I used was to mark the rhythm prior to and after PVC and transfering to R-R. I see no P waves during the episode of (artifact or Afib) leading to my questions. There are many documented cases of (athletic Lone afib) with HR well below 70

Paroxysmal AFib. Is a sudden onset and termination from seconds to days according to Cleveland Clinic.

Fibrillatory waves ( AFIB) line 2. Fine amplitude. This is my question. S is Sinus Thanks.

Thank you. Question: Given the QRS complexes have no artifact to them, could these waves be F-waves with (5-1, 6-1 conduction ) resulting in NSR? I cant make out P waves but I trust your read.

My error. This is single lead event monitor. 32 second continous read . 25mm/sec.

Agree on AFL assessment. Does there appear to be any irregularity to rhythm? I see ni P waves.

Here is an example of what? AF or AFL or Artifact. Note QRS complexes clearly defined. Looks to be SR but possible slightly irregularly irregular? Also PACs and PVCs in ECG. Thanks for feedback! Shutterfly