i am sooo sorry for your loss.
. group b meningitis is rare... but it does occur. women should be tested prior to delivery and given antibiotics. i was gbs positive for both of my pregnancy. even though i was treated......i lived in constant fear those first few weeks of their of their little lives (they are 15 and 16 now).
of the 50% of infants born to gbs moms will be colonized. only 1-2% of those colonized infants will become ill. late gbs onset is one week to 90 days. once infected they become septic, and the mortality is high. these infants become critically ill quickly and their immature immune systems cannot fight the infection.
i have worked in emergency medicine for many years and gbs is one of the things we test for in a neonate (<90 days old) with fever in a septic work up. treatment with antibiotics during delivery has greatly reduced the incidence of this disease, but it still occurs.
it is no ones "fault", especially not yours,
that this horrible event happened to your family. even when the mother is treated with antibiotics at delivery, the baby is watched closely, and it is discussed at every well baby visit......will not prevent gbs infection.
i am so sorry this happened to you. i will pray for your heart,and your families hearts to heal.
medscape: medscape access
group b streptococcus (gbs or streptococcus agalactiae
) is an encapsulated gram-positive diplococcus that colonizes the gastrointestinal and genital tracts of 15 to 40 percent of pregnant women. gbs colonization is usually asymptomatic. however, maternal colonization is the primary risk factor for gbs infection in neonates and young infants (younger than 90 days of age). vertical transmission generally occurs after the onset of labor or rupture of the fetal membranes.
group b streptococcus
, also known as streptococcus agalactiae,
was once considered a pathogen of only domestic animals, causing mastitis in cows. s agalactiae
is now best known as a cause of postpartum infection and as the most common cause of neonatal sepsis
. more recently, numerous series have described s agalactiae
as a cause of infection in nonpregnant adults, providing descriptions of the clinical spectrum of disease, including clinical features, risk factors, therapy, and outcome of group b streptococcal infection in nonpregnant adults.
group b streptococci colonize the vaginal and gastrointestinal tracts in healthy women, with carriage rates ranging from 15%-45%. neonates can acquire the organism vertically in utero or during delivery from the maternal genital tract. although the transmission rate from mothers colonized with s agalactiae
to neonates delivered vaginally is approximately 50%, only 1-2% of colonized neonates go on to develop invasive group b streptococcal disease.
neonatal group b streptococcal disease is divided into early and late disease. early group b streptococcal neonatal sepsis often presents within 24 hours of delivery but can become apparent up to 7 days postpartum. no specific clinical features differentiate early group b streptococcal disease from that caused by other pathogens. pneumonia with bacteremia is common, while meningitis is less likely.
late group b streptococcal neonatal sepsis is defined as infection that presents between one week postpartum and age 3 months
. late disease commonly involves group b streptococcus
serotype iii, typically characterized by bacteremia and meningitis.
the absence of antibody to group b streptococci in infants is a risk factor for infection.
i love medscape.
these following links will lead to medscape. medscape requires registration but it is free and is th best source/esource for information.
[color=#1122cc]neonatal meningitis treatment & management
[color=#1122cc]streptococcus group b infections
[color=#1122cc]streptococcus group b infections treatment & management
[color=#1122cc]group b strep - meningitis foundation of america