I'm not the most knowledgable person on all these topics, but I believe (and Merck's manual http://www.merckmanuals.com/professi....html#CIHCIAFF
backs me up) that:
1) Vomiting is a CAUSE of metabolic alkalosis, not a symptom. When you suddenly lose your stomach contents, especially repeatedly, your stomach's parietal cells go hey, too little acid here, I must make more! and more,,,,and more...as the vomiting repeats. And what is a byproduct of stomach acid production? Bicarb ions, which then build up in the rest of your body faster than you can get rid of them. Then you have metabolic alkalosis.
2) A big s/sx of metabolic alkalosis I didn't see you mention was hypoventilation
. Your body is metabolically less motivated than usual to blow off CO2, because the trigger for letting go of CO2 is set off by the acidic end of your acid/base balance, so the urge to breathe comes less frequently, and your respiratory rate is slower.
When I took A&P a few years ago, the textbook said that you could tell different forms of acidosis/alkalosis apart by how they were being compensated. If it was a metabolic imbalance, it would be most visibly compensated through the respiratory system. If it was respiratory imbalance, it would be compensated most visibly by metabolic routes.
Also, alkalosis in all its forms is relatively rare since the causes of alkalosis happen less frequently.
Does all that help?
And now I have a question of my own: I remember one professor saying that our bicarb buffer system buffers Xes better than Y's but I forget which one of those was acids and which one was bases. And, just through a 10 minute internet search, I couldn't puzzle out which one it was. I think it has to do with the relative concentrations of all substances in that equilibrium reaction in the body. Anyone know?